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Client Publication | Nature Communications | Xiangya Second Hospital team led by Dr. Ruping Dai and Dr. Hui Li reveals the hypothalamic paraventricular nucleus CRH neurons’ regulatory pathway in acute lung injury

Scientists from Xiangya Second Hospital reveal how hypothalamic CRH neurons regulate the sympathetic–neutrophil axis to protect against acute lung injury.

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Breakthrough! Chinese Team Publishes in Cell: Gene-Edited Stem Cells Rejuvenate Aging Monkeys

Chinese researchers developed FOXO3 gene-edited stem cells that reversed aging in primates. The study, published in Cell, reveals exosome-mediated systemic rejuvenation.

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Client Publication | Nature Communications | Collaborative Study by Prof. Zhong Chen (Zhejiang Chinese Medical University) and Academician Qingming Luo (Hainan University) Maps Long-Range Inputs to Mouse Histaminergic Neurons

On August 29, the research team led by Prof. Zhong Chen and Qingming Luo published “A whole-brain male mouse atlas of long-range inputs to histaminergic neurons.”

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Client Publication | Neuron | Collaborative Discovery by Dr. Juan Song’s Team Reveals Distinct Circuit Targets for Cognitive and Emotional Deficits in Alzheimer’s Disease

Dr. Juan Song’s team published “Segregated supramammillary–dentate gyrus circuits modulate cognitive and affective function in healthy and Alzheimer’s disease model mice.”

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CUSTOMER SUPPORT

Client Publication | Nature Communications | Xiangya Second Hospital team led by Dr. Ruping Dai and Dr. Hui Li reveals the hypothalamic paraventricular nucleus CRH neurons’ regulatory pathway in acute lung injury

Breakthrough! Chinese Team Publishes in Cell: Gene-Edited Stem Cells Rejuvenate Aging Monkeys

Client Publication | Nature Communications | Collaborative Study by Prof. Zhong Chen (Zhejiang Chinese Medical University) and Academician Qingming Luo (Hainan University) Maps Long-Range Inputs to Mouse Histaminergic Neurons

Client Publication | Neuron | Collaborative Discovery by Dr. Juan Song’s Team Reveals Distinct Circuit Targets for Cognitive and Emotional Deficits in Alzheimer’s Disease

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